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Issue title: Selected Proceedings of the 16th Conference of the European Society for Clinical Hemorheology and Microcirculation (ESCHM), 18–21 June, 2011, Munich, Germany
Article type: Research Article
Authors: Forconi, Sandro | Wild, Philip | Munzel, Thomas | Gori, Tommaso
Affiliations: Centro Siena-Toronto, University of Siena, Siena, Italy | Medical Clinic and Policlinic, University Medical Center Mainz, Mainz, Germany
Note: [] These authors contributed equally to this paper.
Note: [] These authors contributed equally to this paper.
Note: [] Corresponding author: Tommaso Gori, MD, PhD, 2. Medical Clinic and Policlinic, University Medical Center Mainz, Mainz, Germany. E-mail: Tommaso.gori@unimedizin-mainz.de
Abstract: The role of viscosity, and of interindividual variations in this parameter, in the pathophysiology of cardiovascular disease remain incompletely understood. Any speculation regarding the possible impact of “hemorheological” therapies is therefore even more complex. In the last years, the debate regarding the relationship between increased viscosity and atherogenesis has been opened again. While the traditional view postulates that an increased blood viscosity has invariably a negative impact on tissue perfusion and therefore should be considered as a risk factor (when not as a true disease), a more recent hypothesis has been formulated based on the observation that small increases in viscosity actually have vasodilatory effects, potentially improving tissue perfusion.
Keywords: Coronary slow flow, endothelial function, viscosity, hematocrit, platelets
DOI: 10.3233/CH-2011-1498
Journal: Clinical Hemorheology and Microcirculation, vol. 49, no. 1-4, pp. 487-491, 2011
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